Displaying items by tag: attenuation
Sunday, 27 December 2015 16:48
14262
hs/ke/nl/rg.
Date : 00.00.00
Name of the Patient : Abc XyzJalmn / M / 65 yrs.
Referred by : Dr. Abc Xyzbar.
Examination : Intracranial and Neck M.R.A.
CLINICAL PROFILE :
C/O gait ataxia and diplopia.
Known hypertensive.
EXAMINATION :
The brain was screened with 5 mm thick T2 Weighted axial images.
Intracranial and neck MRA were performed with 3D TOF and 2D TOF sequences, respectively.
OBSERVATION :
There are small areas of hyperintensity on the T2 Weighted images within the right thalamus (se/im: 102/11) and in the left centrum semiovale (se/im: 102/15) and these are most likely ischemic in etiology.
There is mild fullness of the third and both the lateral ventricles. There is mild prominence of the cerebral cortical sulci bilaterally.
Note is made of an empty sella. Also seen is slight thinning of the corpus callosum.
Prominent perivascular spaces are noted within both lentiform nulcei.
The fourth ventricle is normal. There is no shift of the midline structures.
INTRACRANIAL MRA :
There is attenuation of the flow signal within the right anterior cerebral artery and right posterior cerebral artery. Also seen is slight attenuation in the left posterior cerebral artery and this may be due to atherosclerosis.
..2/.
The petrous, cavernous and supraclinoid segments of the internal carotid arteries bilaterally show normal signal and calibre. The visualized left anterior cerebral, middle cerebral, basilar, vertebral arteries also show normal signal, calibre and wall margins. No obvious aneurysm or vascular malformation is identified.
NECK MRA :
The common carotid arteries and their extracranial branches appear normal bilaterally. There are no vessel wall irregularities or stenosis of the vessels noted.
IMPRESSION :
1. Areas of altered signal within the right thalamus and in the left centrum semiovale are most likely ischemic in etiology.
2. Attenuation of the flow signal within the right anterior cerebral artery and right posterior cerebral artery and slightly in the left posterior cerebral artery, this may be due to atherosclerosis.
Date : 00.00.00
Name of the Patient : Abc XyzJalmn / M / 65 yrs.
Referred by : Dr. Abc Xyzbar.
Examination : Intracranial and Neck M.R.A.
CLINICAL PROFILE :
C/O gait ataxia and diplopia.
Known hypertensive.
EXAMINATION :
The brain was screened with 5 mm thick T2 Weighted axial images.
Intracranial and neck MRA were performed with 3D TOF and 2D TOF sequences, respectively.
OBSERVATION :
There are small areas of hyperintensity on the T2 Weighted images within the right thalamus (se/im: 102/11) and in the left centrum semiovale (se/im: 102/15) and these are most likely ischemic in etiology.
There is mild fullness of the third and both the lateral ventricles. There is mild prominence of the cerebral cortical sulci bilaterally.
Note is made of an empty sella. Also seen is slight thinning of the corpus callosum.
Prominent perivascular spaces are noted within both lentiform nulcei.
The fourth ventricle is normal. There is no shift of the midline structures.
INTRACRANIAL MRA :
There is attenuation of the flow signal within the right anterior cerebral artery and right posterior cerebral artery. Also seen is slight attenuation in the left posterior cerebral artery and this may be due to atherosclerosis.
..2/.
The petrous, cavernous and supraclinoid segments of the internal carotid arteries bilaterally show normal signal and calibre. The visualized left anterior cerebral, middle cerebral, basilar, vertebral arteries also show normal signal, calibre and wall margins. No obvious aneurysm or vascular malformation is identified.
NECK MRA :
The common carotid arteries and their extracranial branches appear normal bilaterally. There are no vessel wall irregularities or stenosis of the vessels noted.
IMPRESSION :
1. Areas of altered signal within the right thalamus and in the left centrum semiovale are most likely ischemic in etiology.
2. Attenuation of the flow signal within the right anterior cerebral artery and right posterior cerebral artery and slightly in the left posterior cerebral artery, this may be due to atherosclerosis.
Published in
MRI Reports