sb/hs/rg/nl
Date : 00.00.00

Name of the Patient : Abc XyzSalmn / M / 85 yrs.
Referred by : Dr. Abc Xyzah.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

C/O seizures since 5 years.
H/O fall with loss of consciousness on 00.00.00.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.
5 mm thick FLAIR and Fast Scan (T2 *) coronal images.
SOME IMAGES SHOW PATIENT MOTION.

OBSERVATION :

There is seen a small hypointense focus on the T1 Weighted images in the left cerebellar hemisphere, along the left lateral margin of the fourth ventricle. This lesion appears hyperintense with a hypointense rim on the proton and T2 Weighted images but is significantly hypointense on the Fast Scan (T2 *) images.

There are ill-defined, hyperintense areas on the proton, T2 Weighted and FLAIR images in the subcortical white matter in the left fronto-parietal region, periventricular white matter bilaterally and in the subcortical white matter in the right posterior parietal region. These lesoins appear iso to hypointense to normal white matter on the T1 Weighted images and most likely represents ischemic changes.

Lacunar infarcts are noted in the right cerebellar hemisphere and right external capsular region.

There is mild dilatation of both the lateral and the third ventricles. The fourth ventricle is normal.

- 2 - Scan-00002

There is slight prominence of the cerebral cortical sulci, cerebellar folia and basal cisternal spaces bilaterally. There is no shift of the midline structures. Ectasia of the vertebro-basilar and carotid vessels is noted.

Ill-defined, hyperintense signal seen on the T2 Weighted and Fast Scan (T2 *) images in the subgaleal soft tissues in the right fronto-parietal region and in the left high parietal region may represent subgaleal contusions in the given clinical setting.

Inflammatory changes are noted in the ethmoidal air cells bilaterally.

IMPRESSION :

1. Altered signal intensity lesion in the left cerebellar hemisphere, along the left lateral margin of the fourth ventricle follows the signal characteristics of hemosiderin and may be the sequelae of a previous hematoma.

2. Altered signal in the subcortical white matter in the left fronto-parietal region, periventricular white matter bilaterally and in the subcortical white matter in the right posterior parietal region most likely represent ischemic lesions.

3. Lacunar infarcts in the right cerebellar hemisphere and right external capsular region.

4. Altered signal in the subgaleal soft tissues in the right fronto-parietal region and in the left high parietal region may represent subgaleal contusions in the given clinical setting.

5. Cerebral cortical and cerebellar atrophy.

As compared to the previous MRI dated 00.00.00 (scan nos.0000/5), except for the subgaleal contusions as described, there is no other significant change noted.