sb/ke/nl/rg.
Date : 00.00.00

Name of the Patient : Abc Xyz S. Bhlmn / M / 65 yrs.
Referred by : Dr. Abc Xyzbar.
Examination : M.R.I. of the Brain & I.A.M.

CLINICAL PROFILE :

C/O tingling on the left side of the body since 00.00.00.
Known diabetic/hypertensive.

EXAMINATION :

M.R.I of the brain and I.A.M. was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

3 mm thick T1 Weighted coronal images.

MR cisternogram was obtained in the coronal plane.

5 mm thick T1 Weighted sagittal images.

OBSERVATION :

There is seen an approximately 1.0 x 1.0 x 1.0 cms sized well-defined, intermediate signal intensity mass lesion on the T1 Weighted images in the left cerebello-pontine angle cistern. This lesion appears relatively hypointense on the T2 Weighted images and is seen to extend into the left internal auditory canal along the left seventh and eighth cranial nerve complex.

There are ill-defined, hyperintense areas on the proton and T2 Weighted images in the posterior parietal, paraventricular white matter bilaterally and in the subcortical and frontal deep white matter bilaterally. These lesions appear iso to hypointense to normal white matter on the T1 Weighted images.

A lacunar infarct is noted in the left lentiform nucleus.


There is mild dilatation of both the lateral and third ventricles. The fourth ventricle is normal. There is prominence of the cerebral cortical sulci, cerebellar folia and basal cisternal spaces bilaterally. There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.

Inflammatory changes are noted in the sphenoid sinus.

IMPRESSION :

1. An approximately 1.0 x 1.0 x 1.0 cms sized mass lesion in the left cerebello-pontine angle cistern extending into the left internal auditory canal, most likely represents an acoustic neurinoma.

2. Altered signal in the posterior parietal, paraventricular white matter bilaterally and in the subcortical and frontal deep white matter bilaterally represent ischemic changes.

3. Mild age related cerebral cortical and cerebellar atrophy.