sb/ke/nl/rg.
Date : 00.00.00

Name of the Patient : Abc Xyzisha Ilmn / F / 30 yrs.
Referred by : Dr. Abc Xyzah.
Examination : M.R.I. of the Sella & Perisellar Region.

CLINICAL PROFILE :

C/O loss of vision of both eyes since 1 1/2 months.

EXAMINATION :

M.R.I of the sella and perisellar region was performed using the following parameters :

3 mm thick T1 Weighted, T2 Weighted and STIR coronal images.

3 mm thick T1 Weighted sagittal images.

MR cisternogram was obtained in the sagittal plane.

The brain was screened with 5 mm thick T2 Weighted axial images.

OBSERVATION :

There is seen an approximately 2.0 x 1.7 x 2.3 cms sized well marginated, lobulated, intermediate signal intensity mass lesion on the T1 Weighted images in the sella and suprasellar region. This lesion appears relatively hypointense on the T2 Weighted images. The pituitary stalk is not well identified on this study. The optic chiasma and the proximal optic nerves are not well identified separately from the lesion. Effacement of the suprasellar cistern is noted. Probable extension of the lesion into the right cavernous sinus is seen with encasement of the cavernous segment of the right internal carotid artery.

The posterior pituitary gland reveals normal hyperintense signal on the T1 Weighted images. The hypothalamus is unremarkable.



Inflammatory changes are noted in the sphenoid sinus on the left.

Screening images of the brain reveal hyperintense signal on the T2 Weighted images in the frontal deep white matter bilaterally and in the body of the corpus callosum anteriorly.
There is mild fullness of both the lateral ventricles. The third and the fourth ventricles are normal. There is no midline shift.

The optic nerves bilaterally show normal signal intensity.

IMPRESSION :

1. An approximately 2.0 x 1.7 x 2.3 cms sized lobulated, mass lesion in the sella and suprasellar region most likely represents a pituitary macroadenoma.

A contrast enhanced scan may be worthwhile.

2. Altered signal in the frontal deep white matter bilaterally and in the body of corpus callosum anteriorly as described is not specific for a single etiology. These may represent ischemic lesions or demyelinating plaques.