Sunday, 27 December 2015 16:48

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sb/hs/nl/nl
Date : 00.00.00

Name of the Patient : Abc Xyzr Kulklmn / M / 81 yrs.
Referred by : Dr. Abc Xyzah.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

H/O seizures since 0000. On anti-epileptics.
Last episode on 30th April 0000.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

5 mm thick FLAIR coronal images.

3 mm thick T1 Weighted and T2 Weighted coronal images.

OBSERVATION :

There are ill-defined hyperintense areas on the proton, T2 Weighted and FLAIR images in the posterior parietal periventricular white matter bilaterally and in the subcortical white matter in the frontal and parietal regions bilaterally. These lesions appear iso to hypointense to normal white matter on the T1 Weighted images.

Lacunar infarcts (iso to hyperintense to CSF) are noted in the left cerebellar hemisphere and in the thalami bilaterally.

The hippocampal complex is unremarkable on either side.

There is mild dilatation of both the lateral and third ventricles. The fourth ventricle is normal. There is prominence of the cerebral cortical sulci, cerebellar folia and basal cisternal spaces bilaterally.
Scan-00009


There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.

Inflammatory changes are noted in the mastoid air cells bilaterally and in the right maxillary antrum.

IMPRESSION :

1. Altered signal in the posterior parietal periventricular white matter bilaterally and in the subcortical white matter in the frontal and parietal regions bilaterally most likely represent ischemic changes.

2. Lacunar infarcts in the left cerebellar hemisphere and in the thalami bilaterally.

3. Age related cerebral cortical and cerebellar atrophy.
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