sb/ke/nl/nl
Date : 00.00.00

Name of the Patient : Abc Xyz Dlmn / M / 62 yrs.
Referred by : Dr. Abc Xyzah.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

C/O irrelevant talk since 8 days.
Known hypertensive.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

5 mm thick FLAIR coronal images.

OBSERVATION :

There is a wedge-shaped hypointense lesion on the T1 Weighted images involving the cortex and the subcortical white matter in the left temporo-parietal region. This lesion appears hyperintense on the proton, T2 Weighted and FLAIR images. Ill-defined, hyperintense signal on the T1 Weighted images within this lesion, which appears relatively hypointense on the T2 Weighted images representing intracellular methemoglobin and suggests early subacute haemorrhage. There is resultant effacement of the sulcal spaces in the left temporo-parietal region.

There is a CSF intensity lesion on all the pulse sequences in the right occipital lobe, which represents an area of cystic encephalomalacia, most likely the sequelae of a previous vascular insult. Perilesional, white matter hyperintense signal on the T2 Weighted and FLAIR images may represent gliotic changes.






Ill-defined hyperintense areas on the proton, T2 Weighted and FLAIR images in the pons and bilateral centrum semiovale, most likely represent ischemic changes.

Lacunar infarcts are noted in the pons, genu of the corpus callosum on the left, right thalamus and bilateral lentiform nuclei.

There is mild dilatation of the ventricular system. There is prominence of the cerebral cortical sulci and cerebellar folia.

The basal cisternal spaces are unremarkable. There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.

IMPRESSION :

1. A haemorrhagic infarct in the left temporo-parietal region.

2. An area of cystic encephalomalacia with perilesional gliosis in the right occipital lobe, most likely the sequelae of previous vascular insult.

3. Altered signal in the pons and bilateral centrum semiovale, most likely represent ischemic changes.

4. Lacunar infarcts in the pons, genu of the corpus callosum on the left, right thalamus and bilateral lentiform nuclei.