sb/ke/rg.
Date : 00.00.00

Name of the Patient : Abc Xyz Ylmn / F / 40 yrs.
Referred by : Dr. Abc Xyzshar / Dr. Abc Xyzri.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

C/O tingling in the LUE and LLE since May 0000.
For follow up.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

5 mm thick FLAIR coronal images.

Limited 2D PC MR Angiogram images were obtained in the axial and coronal planes.

OBSERVATION :

There is an ill-defined, predominantly hypointense signal on the T1 Weighted images in the right high parietal cortex. This lesion appears hyperintense on the T2 Weighted and FLAIR images and represents gliotic/encephalomalacic changes, the sequelae of a previous vascular insult. Ill-defined, hyperintense signal on all pulse sequences is noted within the above described lesion, which represents extracellular methaemoglobin (subacute haemorrhage).

There is an ill-defined, hypointense signal on the T1 Weighted images along the right fronto-temporo-parietal cortex and subcortical white matter. This lesion appears hyperintense on the proton, T2 Weighted and FLAIR images. Resultant slight effacement of the sulcal spaces in these regions is noted.
scan-00005

The petrous and the cavernous segments of the right internal carotid artery show an intraluminal, hyperintense signal on all pulse sequences, instead of the normal flow-void signal. On the MRA sequences, there is non-visualization of the right internal carotid artery.

Both the lateral, third and the fourth ventricles are normal. The basal cisternal spaces are unremarkable. There is no shift of the midline structures.

IMPRESSION :

1. Alterd signal in the right high parietal cortex represents gliotic/encephalomalacic changes, the sequelae of a previous vascular insult. Evidence of subacute haemorrhage is also noted.

2. Altered signal along the right fronto-temporo-parietal cortex and subcortical white matter follows the signal characteristics of a recent ischemic lesion.

3. Slow flow/thrombus in the petrous and cavernous segments of the right internal carotid artery with non-visualization of the intracranial segment of the right internal carotid artery.

As compared to the previous limited MRI dated 00.00.00 (scan no.00006), there is an increase in the extent of the lesion. There is also evidence of slow flow/thrombus in the petrous and cavernous segments of the right internal carotid artery on the present study.