ke/sb/nl/nl
Date : 00.00.00

Name of the Patient : Abc Xyzudan lmn / M / 82 yrs.
Referred by : Dr. Abc Xyzmpat.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

H/O right sided hemiparesis (1st in 0000 and 2nd in 0000). Recovered.
C/O altered sensorium since 3 days.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

5 mm thick FLAIR and Fast Scan (T2 *) coronal images.

OBSERVATION :

There is an area in the left parieto-occipital region which is seen to follow CSF signal intensity characteristics on all the pulse sequences. Hyperintense areas are noted on the proton, T2 Weighted and FLAIR images at the periphery of this lesion which would represent gliosis. There is dilatation of the occipital horn and atrium of the left lateral ventricle. This lesion would represent an area of cystic encephalomalacia.

Hyperintense areas are seen on the proton, T2 Weighted and FLAIR images in the pons, right thalamus and the periventricular deep white matter. These are hypointense to normal white matter on the T1 Weighted images and are suggestive of areas of ischemia/infarction. Similar areas are noted in bilateral corona radiata and bilateral centrum semiovale.








Lacunar infarcts and prominent perivascular spaces are seen in the lentiform nuclei and head of the caudate nuclei bilaterally and the left thalamus.

There is mild dilatation of both the lateral and third ventricles. The fourth ventricle is normal. There is prominence of the cerebral cortical sulci, Sylvian fissures, basal cisterns and the cerebellar folia bilaterally.

There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.

IMPRESSION :

1. Areas of cystic encephalomalacia in the left parieto-occipital region, the sequelae of previous vascular insult.

2. Altered signal in the pons, right thalamus, periventricular deep white matter, bilateral corona radiata and bilateral centrum semiovale are suggestive of areas of ischemia/ infarction.

3. Cerebral and cerebellar atrophy.