sb/ke/nl/rg.
Date : 00.00.00

Name of the Patient : Abc Xyzen lmn / F / 75 yrs.
Referred by : Dr. Abc Xyzrikh.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

C/O loss of speech from 5.00 pm on 00.00.00 with 1 episode of vomiting and loss of consciousness.
H/O headaches on the same day.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

5 mm thick FLAIR and Fast Scan (T2 *) coronal images.

OBSERVATION :

There is an ill-defined, hyperintense signal on the proton, T2 Weighted and FLAIR images in the cerebellar hemispheres bilaterally and at the right occipital pole. This lesion appears hypointense to normal white matter on the T1 Weighted images and represents a recent infarct. Resultant effacement of the cerebellar folia is noted bilaterally.

There are ill-defined, hyperintense areas on the proton, T2 Weighted and FLAIR images in the subcortical white matter in the fronto-parietal regions bilaterally, posterior parietal periventricular white matter bilaterally and in the left thalamus. These appear iso to hypointense to white matter on the T1 Weighted images and represent ischemic changes.







There is mild fullness of both the lateral ventricles. The third and the fourth ventricles are normal. There is slight prominence of the cerebral cortical sulci bilaterally. The basal cisternal spaces are unremarkable. There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.

There is no evidence of haemorrhage on this study.

IMPRESSION :

1. Non-haemorrhagic, recent infarcts in the cerebellar hemispheres bilaterally and at the right occipital pole.

2. Altered signal in the subcortical white matter in the fronto-parietal regions bilaterally, posterior parietal periventricular white matter bilaterally and in the left thalamus represent ischemic changes.