sb/hs/rg.
Date : 00.00.00

Name of the Patient : Abc Xyzandra lmn / M / 56 yrs.
Referred by : Dr. Abc Xyzheja / Dr. Abc Xyzauhan.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

C/O right sided hemiparesis with slurred speech, drooping of the left eyelid and diplopia since 00.00.00
Known diabetic.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.
4 mm thick FLAIR coronal images.
3 mm thick T1 Weighted and STIR coronal images through the cavernous sinus and the orbits.

OBSERVATION :

There is an ill-defined hyperintense signal on the proton, T2 Weighted and FLAIR images in the left cerebral peduncle and in the midbrain, in the midline anteriorly. This lesion appears hypointense to normal white matter on the T1 Weighted images and most likely represents ischemic lesions, probably recent in the given clinical setting.

There are ill-defined, hyperintense areas on the proton, T2 Weighted and FLAIR images in the posterior parietal periventricular white matter on the left and in the subcortical white matter in the fronto-parietal regions bilaterally. These lesions appear iso to hypointense to white matter on the T1 Weighted images and these also most likely represent ischemic lesions.

There is mild fullness of both the lateral ventricles. The third and the fourth ventricles are normal. The basal cisternal spaces are unremarkable. There is no shift of the midline structures. R>
Hyperintense signal is seen within the left sigmoid and transverse sinuses (? slow flow).

The cavernous sinuses, sella and the suprasellar cistern
s are unremarkable on either side. The optic nerves on either side appear normal in course and signal characteristics.

Inflammatory changes are noted in the left mastoid air cells.

IMPRESSION :

The MRI features are suggestive of :

1. Altered signal in the left cerebral peduncle and in the midbrain, in the midline anteriorly most likely represents ischemic lesions, probably recent in the given clinical setting.

2. Altered signal in the posterior parietal periventricular white matter on the left and in the subcortical white matter in the fronto-parietal regions bilaterally are
also most likely ischemic in etiology.

3. No abnormality is detected in the region of the cavernous sinuses, sella or suprasellar cisterns on this study.