sb/hs/nl/nl
Date : 00.00.00
Name of the Patient : Abc Xyzn Nalmn / M / 60 yrs.
Referred by : Dr. Abc Xyzni.
Examination : M.R.I. of the Dorso-lumbar Spine.
CLINICAL PROFILE :
Alleged H/O fall 6 months ago with backache and pain radiating to BLE with paresthesias.
EXAMINATION :
M.R.I of the dorso-lumbar spine was performed using the following parameters :
5 mm thick T1 Weighted and T2 Weighted sagittal images.
6 mm thick T1 Weighted and T2 Weighted axial images.
OBSERVATION :
There is central and anterior wedging of the D11, D12 and L1 vertebral bodies. These vertebrae show an ill-defined, hypointense signal on the T1 Weighted images which appears iso to slightly hyperintense to normal marrow on the T2 Weighted images. There is break in the cortical endplates adjacent to the D11-D12 and D12-L1 intervertebral discs. The D12-L1 intervertebral disc is herniated, centrally, into the body of L1 and shows a hyperintense signal on the T2 Weighted images. Minimal retroplacement of the D11 and D12 vertebrae is noted in relation to the rest of the dorso-lumbar vertebrae. Slight involvement of the D11-D12 intervertebral disc is also noted. There is slight extension of this pathologic process into the pre and paravertebral soft tissues at the D11 and D12 vertebral levels.
Small posterior disc bulges are noted at the L3-L4 and L4-L5 levels and a postero-central protruded disc is noted at the L5-S1 level. The L5-S1 facet joints show mild degenerative changes.
The rest of the visualized dorso-lumbar vertebral bodies show preponderance of hematopoeitic marrow (hypointense on the T1 Weighted images). The remaining visualized dorso-lumbar intervertebral discs show slight loss of water content. The rest of the facet joints are unremarkable.
The visualized lower dorsal spinal cord shows normal signal intensity. There is no cord compression.
The conus medullaris terminates at the L1 level.
IMPRESSION :
Central and anterior wedging of the D11, D12 and L1 vertebrae with altered signal as described and herniation of the D12-L1 intervertebral disc into the body of L1, may be the sequelae of previous trauma. An infective lesion cannot be entirely excluded.
The possibility of a neoplastic etiology is less likely.
There is no cord compression or cord signal alteration.