hs/ke
Date : 00.00.00

Name of the Patient : Abc Xyz lmn / F / 65 yrs.
Referred by : Dr. Abc Xyz Mehta.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

C/O loss of speech since 12.30 pm on 00.00.00.
Known hypertensive.
C/O gait imbalance since 4 years.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

5 mm thick FLAIR and Fast Scan (T2 *) coronal images.

OBSERVATION :

There are diffuse irregularly defined areas of hypointensity on the T1 Weighted images which turn hyperintense on the proton, T2 Weighted and FLAIR images within the periventricular white matter, corona radiata and centrum semiovale bilaterally. These are most likely ischemic in etiology.

Smaller areas with similar signal characteristics are noted within the pons and lentiform nuclei bilaterally.

Lacunar infarcts (iso to hyperintense to CSF) are seen within the lentiform nuclei and thalamus bilaterally.

An area of marked hypointensity on the Fast Scan (T2 *) images is seen within the left lentiform nucleus and this would most likely represent hemosiderin the result of previous bleed.






There is fullness of the third and both the lateral ventricles. There is prominence of the cerebral cortical sulci and Sylvian fissures bilaterally. There is slight prominence of the basal cisternal spaces bilaterally.

The fourth ventricle is normal. There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.

IMPRESSION :

1. Areas of altered signal within the periventricular white matter, corona radiata, centrum semiovale, pons and lentiform nuclei bilaterally are most likely ischemic in etiology.

2. Lacunar infarcts within the lentiform nuclei and thalamus bilaterally.

3. Residual hemosiderin, the result of previous bleed within the left lentiform nucleus