Sunday, 27 December 2015 16:48

12004

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Date : 00.00.00

Name of the Patient : Abc Xyzen Sanlmn / F / 74 yrs.
Referred by : Dr. Abc Xyzhah.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

C/O sudden onset of weakness on the left side since 2 days.
Known hypertensive.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

5 mm thick FLAIR and Fast Scan (T2 *) coronal images.

OBSERVATION :

There are ill-defined, hyperintense areas on the proton, T2 Weighted and FLAIR images in the posterior parietal periventricular white matter bilaterally and in the lentiform nuclei, corona radiata and centrum semiovale bilaterally. These lesions appear iso to hypointense to normal white matter on the T1 Weighted images.

A lacunar infarct is noted in the head of the right caudate nucleus.

There is evidence of volume loss in the left posterior parietal and high parietal cortex without change in signal intensity. This may be a sequelae of previous vascular insult.

There is a small hyperintense epidural lesion in the left high parietal region (se/im. 105.12) ? cause ? haemorrhage.

There is mild fullness of both the lateral and third ventricles. The fourth ventricle is normal. There is slight prominence of the cerebral cortical sulci, cerebellar folia and basal cisternal spaces bilaterally. There is no midline shift.
Scan-00004


No obvious vascular anomaly is identified on this study.

There is no obvious haemorrhage on this study.

IMPRESSION :

1. Altered signal in the posterior parietal periventricular white matter bilaterally and in the lentiform nuclei, corona radiata and centrum semiovale bilaterally most likely represent ischemic changes.

2. A lacunar infarct in the head of the right caudate nucleus.

3. Volume loss in the left posterior parietal and high parietal cortex may be a sequelae of previous vascular insult.



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