ke/sb
Date : 00.00.00

Name of the Patient : Abc XyzD. lmn / F / 65 yrs.
Referred by : Dr. Abc Xyzrankar.
Examination : M.R.I. of the Dorso-lumbar Spine.

CLINICAL PROFILE :

H/O backache radiating to BLE with paresthsias since 3-4 months.
C/O weakness of BLE with bladder/bowel involvement since 1 month.

EXAMINATION

M.R.I of the dorso-lumbar spine was performed using the following parameters :

5 mm thick T1 Weighted and T2 Weighted sagittal images.

7 mm thick T1 Weighted and T2 Weighted axial images.

OBSERVATION :

There is central wedging of the L1 and L5 vertebral bodies. The posterior margins of these vertebral bodies are seen to be bulging, posteriorly. Resultant compression of the conus medullaris is noted at the L1 vertebral level. The dorsal spinal cord at the L1 level shows a subtle hyperintense signal on the T2 Weighted images which may suggests cord edema/ischemia.

The majority of the vertebrae of the spinal axis appear slightly wedged and show hypointense signal on the T1 Weighted images. On the T2 Weighted images, these vertebrae show a subtle hyperintense signal, when compared to the normal marrow. The posterior elements of these vertebrae are also involved. These vertebrae appear slightly expansile when compared to normal. The visualized intervertebral discs are unremarkable.

Compromise of the lumbar canal is noted at the L1 and L5 vertebral levels.
- 2 - scan-00004

Focal altered signal is also noted in the iliac bones bilaterally and in the visualized skull valut (109.1-2).

There are also seen focal nodular lesions in the lung parenchyma on the left and probably along the ribs in the right hemithorax.

The conus medullaris terminates at the L1 level.

IMPRESSION :

Altered signal along the spinal axis involving the majority of the vertebral bodies and their posterior elements as described, with focal lesions in the iliac bones, skull valut and in the thorax as described is not specific for a single etiology. These features may represent,

1. Multiple metastasis.

2. Round cell tumor.

3. Less likely to represent an infective etiology.

There is cord compression at the L1 level with cord signal alteration suggesting cord edema/ischemia.