Sunday, 27 December 2015 16:48

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Date : 00.00.00

Name of the Patient : Abc XyzC. Sarvlmn / F / 58 yrs.
Referred by : Dr. Abc Xyzah.
Examination : M.R.I. of the Brain.

CLINICAL PROFILE :

C/O headaches with diminished vision on the right side and memory lapses since 5-6 months.
Known hypertensive. On Rx.

EXAMINATION :

M.R.I of the brain was performed using the following parameters :

5 mm thick T1 Weighted, proton and T2 Weighted axial images.

5 mm thick FLAIR coronal images.

OBSERVATION :

There is volume loss in the left occipital lobe. There is an ill-defined, hypointense lesion on the T1 Weighted images in the left occipital lobe. This lesion follows CSF signal on all pulse sequences. Perilesional white matter hyperintense signal on T2 Weighted and FLAIR images is noted. Mild dilatation of the atrium and occipital horn of the left lateral ventricle is also noted.

There are small bright foci on proton, T2 Weighted and FLAIR images in the deep white matter in the frontal regions bilaterally, right centrum semiovale and in the right frontal periventricular white matter. These lesion appears iso to hypointense to normal white matter on the T1 Weighted images.

Lacunar infarcts are noted in the left lentiform nucleus and left thalamus.

There is mild fullness of both the lateral and the third ventricles. The fourth ventricle is normal. The basal cisternal spaces are unremarkable. There is no shift of the midline structures. No obvious vascular anomaly is identified on this study.
..2/.




- 2 - scan-00006


Inflammatory changes are noted in the maxillary sinuses, sphenoid sinus and right mastoid air cells.

IMPRESSION :

1. Volume loss in the left occipital lobe with altered signal represents an area of cystic encephalomalacia with perilesional gliotic changes most likely the sequelae of a previous vascular insult.

2. Altered signal in the deep white matter in the frontal regions bilaterally, right centrum semiovale and in the right frontal periventricular white matter represents ischemic changes.

3. Lacunar infarcts in the left lentiform nucleus and left thalamus.


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